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Examinando por Autor "Vega Cuesta, Federico"

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    Biosemiótica relacional. Una propuesta de integración de la biología relacional de Rosen y la semiosis de Peirce
    (Universidad Nacional de Educación a Distancia (España). Escuela Internacional de Doctorado. Programa de Doctorado en Filosofía, 2022) Vega Cuesta, Federico; Saborido Alejandro, Cristian
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    The cell as a realization of the (M, R) system
    (Elsevier, 2023-03) Vega Cuesta, Federico; https://orcid.org/0000-0002-8301-7187
    Robert Rosen defines organisms as natural systems closed to efficient causation, and proposes the (M, R) system as a model for them. He argues that the study of this formal system provides an understanding of living beings that cannot be obtained from conventional biology. However, he recognizes that answering specific questions about specific organisms requires identifying his relational concepts with specific characteristics of individual biological systems. Therefore, to apply the proposed model to biological research, it is necessary to recover the material systems through a process of realization of the formal system. The description of the cell as a realization of the (M, R) system has been notoriously problematic to date. This article posits a cell model based on the functional organization of cellular biochemical processes, and on Rosen's construction of the replication function. Then, based on the proposed model, the meaning of the replication function is discussed and the (M, R) system is analyzed as a theory of life.
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    The cell as a semiotic system that realizes closure to efficient causation: The semiotic (M, R) system
    (Elsevier, 2024-06) Vega Cuesta, Federico
    Robert Rosen defines organisms as material systems closed to efficient causation, and proposes the replicative (M, R) system as a model for them. Vega (2023) proposes a cell model that realizes Rosen’s formal model, based on Hofmeyr's (2017) analysis of the functional organization of cell biochemistry and on Rosen's construction of the replication function. In this article we propose a cell model that, starting from the same biochemical processes, replaces the replication function with a set of semiotic relations between some of the elements that participate in cellular processes. The result is a cell model that constitutes a semiotic system that realizes closure to efficient causation: a semiotic (M, R) system. We compare the models of closure that correspond to the replicative (M, R) system and the semiotic (M, R) system. Additionally, we discuss the role that the genetic code and protein synthesis play in the semiotic closure to efficient causation. Finally, we outline the method to extend this analysis to more complex organisms.
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